Traditionally, platelets were considered inert materials that cleverer cells used to make blood clots, as cement is used by architects and builders. Now it is clear that they are active mediators of thrombus formation, and central in the pathogenesis of acute ischemic syndromes including heart attacks and strokes. There has been no talk of reparations, but scientists are being more careful now what they say when platelets might hear. Here researchers and practitioners in cardiovascular medicine discuss such topics as thrombaxane antagonists, monitoring anti-platelet therapy, and future strategies for developing anti-platelet drugs. Distributed in the US by World Scientific.
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